Hmgb1 Knock Out MEF Large T antigen

The elucidation of the anti-inflammatory mechanism of EMO in rheumatoid arthritis through an integrative approach combining bioinformatics and experimental verification
High-mobility group box-1 peptide ameliorates bronchopulmonary dysplasia by suppressing inflammation and fibrosis in a mouse model
MIR-107/HMGB1/FGF-2 axis responds to excessive mechanical stretch to promote rapid repair of vascular endothelial cells
Nucleosome loss facilitates the chemotactic response of macrophages
Ligand-activated peroxisome proliferator-activated receptor-δ and -γ inhibit lipopolysaccharide-primed release of high mobility group box 1 through upregulation of SIRT1
Cross-reactivity of anti-HMGB1 antibodies for HMGB2
Chromatin conformation regulates the coordination between DNA replication and transcription
Intestinal epithelial HMGB1 inhibits bacterial infection via STAT3 regulation of autophagy
FSHD muscle shows perturbation in fibroadipogenic progenitor cells, mitochondrial function and alternative splicing independently of inflammation
A Review of Proposed Mechanisms in Rheumatoid Arthritis and Therapeutic Strategies for the Disease
Blockade of HMGB1 Reduces Inflammation and Pruritus in Atopic Dermatitis by Inhibiting Skin Fibroblasts Activation
NOD2 attenuates osteoarthritis via reprogramming the activation of synovial macrophages
The elucidation of the anti-inflammatory mechanism of EMO in rheumatoid arthritis through an integrative approach combining bioinformatics and experimental verification
Nucleotide-Binding Oligomerization Domain-Like Receptor 3 Deficiency Attenuated Isoproterenol-Induced Cardiac Fibrosis via Reactive Oxygen Species/High Mobility Group Box 1 Protein Axis
Extracellular HMGB1 Contributes to the Chronic Cardiac Allograft Vasculopathy/Fibrosis by Modulating TGF-β1 Signaling
Damage associated molecular pattern molecule-induced microRNAs (DAMPmiRs) in human peripheral blood mononuclear cells
Autophagy-dependent secretion: mechanism, factors secreted, and disease implications
HMGB1 modulates the balance between senescence and apoptosis in response to genotoxic stress
The Association of HMGB1 and RAGE Gene Polymorphisms with IgA Vasculitis
HMGB1: A Common Biomarker and Potential Target for TBI, Neuroinflammation, Epilepsy, and Cognitive Dysfunction
The Anti-inflammatory Effects of HMGB1 Blockades in a Mouse Model of Cutaneous Vasculitis
Nonoxid-HMGB1 Attenuates Cognitive Impairment After Traumatic Brain Injury in Rats
Scaffidi et al. Release of chromatin protein HMGB1 by necrotic cells triggers inflammation. Nature 2002, 418: 191